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In Tregs, relatively weak Akt signaling facilitates the nuclear localization of Foxo proteins, enabling their synergistic interaction with Foxp3 [65]

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It has been previously reported that blockage of the PI3K/AKT pathway by PTEN can be a therapeutic target for RA.[20] The present study results demonstrated the inhibitory effect of GSH on p-PI3K and p-AKT protein expression levels, as well as its promoting effect on the activation of PTEN