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Thus, when γ-glutamylcysteine takes the antioxidant functions, GSH utilization may be preserved for iron–sulfur cluster assembly and protein thiol-redox modifications, as recently reported 9
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303 On the other hand, overexpression of Opa1 helps to mitigate prion-induced fragmentation of mitochondrial networks, loss of mitochondrial DNA, and prevents neuron apoptosis
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