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HDAC4 and 5 are required to suppress apoptosis and ferroptosis and maintain muscle mass in obese mice To establish whether HDAC4 and 5 suppress apoptosis and ferroptosis in skeletal muscle in response to lipoxicity in vivo, a bilateral skeletal muscle loss-of-function HDAC4 and 5 model was developed in obese db/db mice using AAV6 vectors (Fig