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Exposure to hyperoxia triggers compensatory mechanisms to counteract excessive ROS production by activating antioxidant defense pathways and regulating mitochondrial function

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This estimate of 71% for the proportion of Cys catabolized by cysteinesulfinate-dependent pathways in intact rats is similar to the estimate of 63% for hepatocytes isolated from rats fed an adequate diet and incubated with 0.2 mM Cys in vitro