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Studies have shown that NMNAT2 plays a key role in axon maintenance and function, with the depletion or loss of active NMNAT2 causing neurite degeneration [11,12]

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BPC 157 might modulate VEGF expression, whereas in vitro, BPC 157, unlike VEGF, does not stimulate angiogenesis [2,6,192,193] as an important part of the particular local and systemic wound healing effects (for review see [1,2,3,4,5,6,7,8,9,10])

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